05-04-2002, 11:21 AM
• Wiedemayer H, Triesch K, Schafer H and Stolke D (2002). Early seizures following non-penetrating traumatic brain injury in adults: risk factors and clinical significance. Brain Inj. 16 (4): 323-30. Summary: BACKGROUND: In the literature dissenting data are obtained about risk factors for early post-traumatic seizures and their impact on outcome. This study was conducted to obtain more information about the clinical significance of early seizures and their possible impact on the treatment of traumatic brain injury. METHODS AND RESULTS: A consecutive series of 1868 adult patients with head injury were analysed retrospectively. Demographic data of the patients, characteristics of the injury, and findings on CT scan were recorded. Risk factors for early post-traumatic seizures were identified using univariate statistics. A multivariate logistic regression was performed to look for interaction of different variables. The impact of early post-traumatic seizures on outcome was examined in an analogous way. Chronic alcohol abuse, subdural haematoma and brain contusion were identified as independent risk factors for early post-traumatic seizures. A significant association of early post-traumatic seizures with an unfavourable outcome was observed, but this effect was small compared to other variables. CONCLUSIONS: Early post-traumatic seizures appear to be an acute reaction of the brain to cortical damage with little independent impact on the management of head injury. Department of Neurosurgery, University of Essen Medical School, Essen, Germany.
"Early post-traumatic seizures appear to be an acute reaction of the brain to cortical damage with little independent impact on the management of head injury."
If it were a reaction of "the brain" (all systems undiscerned), then why would not all people with brain damages have them. Apart from that there are certain sites in the brain reknown for epilepsy, and others not. I have not heard so far yet, that with brain damaged people it are just the sites that are brain damaged then, instead of the more usual with epilesy.
Also, if it were really the brain damage, then why would the epilespy not go off straight after the injury, where the damages might be expected to be highest.
Therefore I regard it differingly.
In my brain epilepsy and what felt after the concussion like slight prestages to it, are with me to do with a rhythm within a certain span.
With me it can be optical a rhythm of a light flickering / pulse within a certain rhythm for the prestages, or tones within that span.
Too fast or too slow does not do. They have to be within the span, that seems to overlay with an internal rhythm.
There come in brain damages, then, as if it is too much for internal systems, can have to do with brain damages, also caused by previous epileptic fits, after which chances for quite a while might be higher for that it gets too much (with me averaged right faster than left),
and when it gets too much, then in my brain goes off what I call a "cramp-pulsing" of internal stuff along, and with each pulse rapidly spreading to other stuff around.
When it spread enough, it seems to also "sector-hop" and I guess then motor systems get involved, and that that is to do with the jerks of limbs.
So it is not the brain damage by itself, that does the epilepsy, or at least not in my brain.
The brain damage "just" makes that "systems overload" is reached faster there.